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Found 3 matching student topics

Displaying 1–3 of 3 results

A 3D mechanosensing platform for senescent osteocytes in aging bone

Project Reference: #3MPQC-NATP2Preferred Start: Late 2026/Early 2027How do our bones sense movement, and why does this ability decline as we age?Osteocytes are specialised cells embedded throughout bone that detect mechanical forces generated during movement and exercise. They form an interconnected cellular network that helps coordinate the continuous renewal and repair of bone.With age, some osteocytes become senescent: damaged cells that remain within the tissue but no longer function normally. Their accumulation may disrupt how bone senses mechanical loading and contribute …

Study level
PhD, Master of Philosophy, Honours
Faculty
Faculty of Health
School
School of Biomedical Sciences
Research centre(s)
Centre for Biomedical Technologies

Engineering 3D osteocyte–tumour microenvironments to study bone metastasis and therapeutic response

Project Reference: #2MPQC-HuECM1Preferred Project Start: Late 2026/Early 2027How do cancer cells communicate with bone cells, and can we recreate these interactions in the laboratory to improve therapy testing?When cancer spreads to bone, tumour cells enter a highly specialised microenvironment containing multiple cell types and a complex extracellular matrix. Osteocytes are embedded throughout this matrix and form an interconnected cellular network that senses and regulates changes within bone. However, their contribution to cancer progression and treatment response remains poorly understood.Conventional two-dimensional …

Study level
PhD, Master of Philosophy, Honours
Faculty
Faculty of Health
School
School of Biomedical Sciences
Research centre(s)
Centre for Biomedical Technologies

Investigating DNA repair mechanisms in aging adult stem cells

When we age the DNA repair systems of our cells become down regulated. This results in reduced DNA repair capacity, enhanced rates of mutation load and may lead to the development of chronic aging-associated diseases including osteoporosis, Alzheimer's and cancer(1). So it is no surprise that genome instability and stem cell exhaustion, which also strongly correlates with the accumulation of DNA damage, are considered hallmarks of aging(2).However, we still lack a clear understanding on how the decrease in DNA repair …

Study level
PhD, Master of Philosophy, Honours
Faculty
Faculty of Health
School
School of Biomedical Sciences

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