Dr Kah Meng Lee
Academic Division,
Research Portfolio,
Central Analytical Research Facility (CARF)
Biography
Kah Meng (Andi) Lee received her BBiomedSc (Hons) and PhD from The University of Queensland, Australia, where she studied the role of synaptic adhesion molecules in development and maintenance of the neuromuscular junction (NMJ) with Associate Professor Peter Noakes and Associate Professor Nickolas Lavidis. She is currently working as a Senior Technologist (Histology) in Central Analytical Research Facility (CARF). She has specialised skill sets in cryo histology, resin histology and imaging.
Personal details
Positions
- Senior Technologist (Histology)
Academic Division,
Research Portfolio,
Central Analytical Research Facility (CARF)
Qualifications
- Doctor of Philosophy (University of Queensland)
- BBiomedSc(Hons) (University of Queensland)
Publications
- Fan, X., Lee, K., Jones, M., Howard, D., Crawford, R. & Prasadam, I. (2022). A technique for preparing undecalcified osteochondral fresh frozen sections for elemental mapping and understanding disease etiology. Histochemistry and Cell Biology, 158(5), 463–469. https://eprints.qut.edu.au/233798
- Ding, Q., Kesavan, K., Lee, K., Wimberger, E., Robertson, T., Gill, M., Power, D., Chang, J., Fard, A., Mar, J., Henderson, R., Heggie, S., McCombe, P., Jeffree, R., Colditz, M., Hilliard, M., Ng, D., Steyn, F., Phillips, W., Wolvetang, E., Ngo, S. & Noakes, P. (2022). Impaired signaling for neuromuscular synaptic maintenance is a feature of Motor Neuron Disease. Acta Neuropathologica Communications, 10. https://eprints.qut.edu.au/233948
- Wixey, J., Sukumar, K., Pretorius, R., Lee, K., Colditz, P., Bjorkman, T. & Chand, K. (2019). Ibuprofen Treatment Reduces the Neuroinflammatory Response and Associated Neuronal and White Matter Impairment in the Growth Restricted Newborn. Frontiers in Physiology, 10. https://eprints.qut.edu.au/233947
- Wixey, J., Lee, K., Miller, S., Goasdoue, K., Colditz, P., Bjorkman, T. & Chand, K. (2019). Neuropathology in intrauterine growth restricted newborn piglets is associated with glial activation and proinflammatory status in the brain. Journal of Neuroinflammation, 16(1). https://eprints.qut.edu.au/233946
- Chand, K., Lee, K., Lee, J., Qiu, H., Willis, E., Lavidis, N., Hilliard, M. & Noakes, P. (2018). Defects in synaptic transmission at the neuromuscular junction precede motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis. FASEB Journal, 32(5), 2676–2689. https://eprints.qut.edu.au/233961
- Wang, H., Lee, J., Lee, K., Woodruff, T. & Noakes, P. (2017). Complement C5a-C5aR1 signalling drives skeletal muscle macrophage recruitment in the hSOD1G93A mouse model of amyotrophic lateral sclerosis. Skeletal Muscle, 7. https://eprints.qut.edu.au/233958
- Lee, K., Chand, K., Hammond, L., Lavidis, N. & Noakes, P. (2017). Functional decline at the aging neuromuscular junction is associated with altered laminin-a4 expression. Aging, 9(3), 880–899. https://eprints.qut.edu.au/233960
- Chand, K., Lee, K., Lavidis, N. & Noakes, P. (2017). Loss of laminin-a4 results in pre- and postsynaptic modifications at the neuromuscular junction. FASEB Journal, 31(4), 1323–1336. https://eprints.qut.edu.au/233955
- Chand, K., Lee, K., Lavidis, N., Rodriguez Valle, M., Ijaz, H., Koehbach, J., Clark, R., Lew-Tabor, A. & Noakes, P. (2016). Tick holocyclotoxins trigger host paralysis by presynaptic inhibition. Scientific Reports, 6. https://eprints.qut.edu.au/233954
- Chand, K., Lee, K., Schenning, M., Lavidis, N. & Noakes, P. (2015). Loss of ß2-laminin Alters Calcium Sensitivity and Voltage Gated Calcium Channel Maturation of Neurotransmission at the Neuromuscular Junction. The Journal of Physiology, 593(1), 245–265. https://eprints.qut.edu.au/233944
QUT ePrints
For more publications by Kah Meng, explore their research in QUT ePrints (our digital repository).